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Presented for research literature review only Papini E et al
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Following treating with protein synthesis inhibitor cycloheximide (CHX), the half-life of ASCT2 was markedly shortened in WT ZDHHC14-expressing cells, whereas catalytically inactive ZDHHC14 mutant (ZDHHC14 C195S ) only led to a slight change and ZDHHC14-knockout cells extended the half-life of ASCT2 (Fig
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